miR-34 miRNAs provide a barrier for somatic cell reprogramming. Choi YJ, Lin CP, Ho JJ, He X, Okada N, Bu P, Zhong Y, Kim SY, Bennett MJ, Chen C, Ozturk A, Hicks GG, Hannon GJ, He L. Nat Cell Biol. 2011 Oct 23; 13(11):1353-60. doi: 10.1038/ncb2366. [PDF]

Molecular dissection of the miR-17-92 cluster's critical dual roles in promoting Th1 responses and preventing inducible Treg differentiation. Jiang S, Li C, Olive V, Lykken E, Feng F, Sevilla J, Wan Y, He L, Li QJ. Blood. 2011 Oct 4 [Epub ahead of print]. [PDF]

miR-19 is a key oncogenic component of mir-17-92 . Olive V *, Bennett MJ*, Walker JC, Ma C, Jiang I, Cordon-Cardo C, Li Q, Lowe SW, Hannon GJ and He L. Genes and Development. 2009 Dec 15;23(24):2839-49. [PDF]

mir17-92, a cluster of miRNAs in the midst of the cancer network. Olive V, Jiang I and He L. International Journal of Biological Chemistry. [PDF]

microRNAs - powerful repression comes from small RNAs. Ma C, Liu Y and He L. Sci China C Life Sci 2009 Apr;52(4):323-30. [PDF]

The miR-17~92 cluster collaborates with the Sonic hedgehog pathway in medulloblastoma development (2009). Uziel T, Karginov FV, Xie S, Parker JS, Wang Y, Gajjar A, He L, Gilbertson R, Hannon G and Roussel MF. Proc Natl Acad Sci 2009 Feb 24;106(8):2812-7. [PDF]

microRNAs join the p53 pathway, another piece of the tumor suppressor puzzle (2007). He L, He X, Lowe SW and Hannon GJ. Nature Review Cancer [PDF]

Guardian¡¯s little helper ¨C the role of microRNAs in the p53 tumor suppressor network (2007). He X, He L and Hannon GJ. Cancer Research.

A microRNA component of the p53 tumour suppressor network.He L, He X, Lim LP, de Stanchina E, Xuan Z, Liang Y, Xue W, Zender L, Magnus J, Ridzon D, Jackson AL, Linsley PS, Chen C, Lowe SW, Cleary MA and Hannon GJ (2007). Nature 447(7148): 1130-4. [PDF]

A microRNA polycistron as a potential human oncogene (2005). He L, Thomson JM, Hemann MT, Hernando-Monge E, Mu D, Goodson S, Powers S, Cordon-Cardo C, Lowe SW, Hannon GJ and Hammond SM. Nature 435(7043):828-33. [PDF]

MicroRNAs: small RNAs with a big role in gene regulation (2004). He L, Hannon GJ. Nature Review Genetics 5(7):522-31. [PDF]

Spongiform encephalopathy caused by defects of an E3 ubiquitin ligase in mahoganoid mice (2003). He L, Lu XY, Jolly AF, Eldridge AG, Watson SJ, Jackson PK, Barsh GS and Gunn TM. Science 299(5607):710-2.

Molecular and phenotypic analysis of Attractin mutant mice (2001).Gunn TM, Inui T, Kitada K, Ito S, Wakamatsu K, He L, Bouley DM, Serikawa T, Barsh GS. Genetics 158(4):1683-95.

A biochemical function for attractin in agouti-induced pigmentation and obesity (2001). He L, Gunn TM, Bouley DM, Lu XY, Watson SJ, Schlossman SF, Duke-Cohan JS, Barsh GS. Nature Genetics. 27(1):40-7.

Biochemical and genetic studies of pigment-type switching (2000). Barsh G, Gunn T, He L, Schlossman S, Duke-Cohan J. Pigment Cell Research 13 Suppl 8:48-53.

Melanocortin 1 receptor variation in the domestic dog (2000). Newton JM, Wilkie AL, He L, Jordan SA, Metallinos DL, Holmes NG, Jackson IJ, Barsh GS. Mammalian Genome 11(1):24-30.

The mouse mahogany locus encodes a transmembrane form of human attractin (1999). Gunn TM, Miller KA, He L, Hyman RW, Davis RW, Azarani A, Schlossman SF, Duke-Cohan JS, Barsh GS. Nature